Angle-closure glaucomas — posterior (pushing) mechanisms other than pupillary block

Authors

  • Syril Dorairaj Einhorn Clinical Research Center, The New York Eye and Ear Infirmary, New York, New York, USA.
  • Yaniv Barkana Glaucoma Unit, Department of Ophthalmology, Assaf Harofeh Medical Center, Zerifin, Israel.
  • Clement CY Tham Department of Ophthalmology and Visual Sciences, The Chinese University of Hong Kong, Hong Kong, China.
  • Robert Ritch Einhorn Clinical Research Center, The New York Eye and Ear Infirmary, New York, New York, USA.

Abstract

Angle-closure is often erroneously considered synony- mous with pupillary block, the most common mecha- nism leading to acute or chronic iridocorneal apposition. However, abnormalities exist in structures and/or forces other than those related to pupillary block and may cause apposition despite a patent iridotomy. A 4-point classifi- cation of these mechanisms has been described, and includes mechanisms originating in the ciliary body, lens, or posterior segment. This review outlines the character- istics of angle-closure specific to each of these 3 levels.

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Published

2007-12-01

How to Cite

1.
Dorairaj S, Barkana Y, Tham CC, Ritch R. Angle-closure glaucomas — posterior (pushing) mechanisms other than pupillary block. Hong Kong J Ophthalmol [Internet]. 2007Dec.1 [cited 2024Apr.24];11(1):16-22. Available from: https://hkjo.hk/index.php/hkjo/article/view/68

Issue

Vol. 11 No. 1 (2007)

Section

Review Articles

License

The Journal has a fully Open Access policy and publishes all articles under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0) licence. For any use other than that permitted by this license, written permission must be obtained from the Journal.

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